Peace Murjuku: These are the results of the histopathological tests of the little Panagiotakis – “They show” low oxygen in the blood

In the final line it seems to be the case of little Panayiotis, from Amaliada, who lost his life last August in the presence of . Newsit.gr presents the results of histopathological examinations. According to exclusive information, in the brain of the 15-month-old child hypoxic-ischemic neuron formation was detected in periventricular areas and areas of the cortex. CORVERSE An image of cellular bleeding with degenerated red blood cells was presented in the lungs, while in the liver D of the hepatocytes (non-specific finding, compatible with hypoxia/ ischaemia). These are, according to medical examiner Dimitris Galenteris, technical advisor to Panagiotakis’ mother, findings compatible with low oxygen in the blood, which can cause death. “The first point concerning hypoxia is changes in the brain where hypoxic – ischemic formation of neurons is described, the second point is cellular bleeding, which is very often found in sudden neonatal death syndrome, but also in suffocating deaths and of course the lesions in the liver, which is also a finding of hypoxia, non-specific, however it is a finding of hypoxia,” said Mr. Galenteris. ADVERSE “There is no clear pathology that explains the presence of hypoxia and justifies the fatal outcome. There are scattered evidence pathologically, however, they cannot justify sudden death and natural hypoxia in the body. However, we will need to wait for the full forensic report to draw safe conclusions,” he added. More specifically, the results of histopathological tests are as follows: Brain -Departments of brain hemispheres of unknown topography, with normal layers of bark, without cytoarchitectonic disorder. -Berminal oedema areas -Sub-ischemic neuronal formation in periventricular areas and areas of the cortex Focal tigrolysis: Image compatible with prolonged hypoxic ischemic brain attack from hours before death approximately. -Isolated neural formations with morphology of the trial of “flaphoid bodies” (negative to silver staining). A single outbreak of perivascular detoxification is identified in the white substance. The vessels of the cerebral parexus appear viscosally. Rare individual outbreaks of perivascular lymphocyte filtration are identified. A few scattered CD8+s are found T-lemphocytes within the choroid grids and the wall of brain vessels in periventricular areas. Clear diagnostic data compatible with viral encephalitis were not identified in the examined incisions. -No evidence of recent or earlier bleeding Heart -In the wall of the right ventricle there are focal amounts of lymphonuclei identified on the epicardium and expanding to the outer myocardial infarction, without meeting the diagnostic criteria of cardiomyopathy. – Sums of lymphonuclei are also recognized within vascular branches in this area. Lungs – Image of pulmonary oedema of a sufficient degree. -The cells are met to a large extent by red blood cells, which are shown faintly in stained U-E and are shown in immunohistochemical staining of glucoforine, with evidence of degeneration: a picture of cellular bleeding with degenerate red blood cells. There is no substantial vascular congestion, no perivascular or pleural bleeding. – Elements of gastric sediment within the bronchial tree without an associated inflammatory reaction with adjacent Gram (-) micro-organisms development sites, most likely postmortem. – No diffuse cellular damage is observed. – Microgranulomatous lymphocyte formations are observed, without necrosis, with increased apoptosis, peribronchial and scattered in the pulmonary parenchyma. The image is partly compatible with granulomatous lymphocyte median pulmonary disease. Rare nuclear formations that raise the suspicion of viral origin (CMV()) are found. -Integration of perilyla lymph nodes Liver -Recognized maintenance of cell architecture, with the presence of outbreaks of extramyel haematosis (peripylaea and within vaginal) as well as focal perilylae agglomerates agglomerations of lymphonuclei with apoptosis elements. -Diffusion of hepatocellular degeneration (non-specific finding, compatible with hypoxia/ ischemia). bowel – Autolytic lesions in the bowel mucosal. -The area of the blind is identified locations of acute autolysis/ necrosis (?) of the mucosal in the presence of large cells with morphology compatible with viral origin. [(Anosohistochemical pigmentation CMV (-)]. -Inflated mesenterial lymph nodes by activating stem centers. – Butt-like abscess without material alterations. – Other organs or parts of organs without essentials for the diagnosis of histopathological alterations. Comments Granulomatous lymphocyte median pulmonary disease, according to the literature, has been associated with some viral infections and immunodeficiencies. Her clinical relevance in infancy appears to be unclear.